Myopathy Archives

A HaloTag-TEV genetic cassette for mechanical phenotyping of proteins from tissues

Disruption of cardiac thin filament assembly arising from a mutation in LMOD2: A novel mechanism of neonatal dilated cardiomyopathy

Fast skeletal muscle troponin activator CK-2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Myosin filament sliding through the Z-disc relates striated muscle fibre structure to function

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Protein phosphatase 5 regulates titin phosphorylation and function at a sarcomere-associated mechanosensor complex in cardiomyocytes

Reversible temporally-specific inhibition of muscle using a light-activated chloride channel

Myosin Head Configurations in Resting and Contracting Murine Skeletal Muscle

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Deleting Full Length Titin Versus the Titin M-Band Region Leads to Differential Mechanosignaling and Cardiac Phenotypes.

Decreased force enhancement in skeletal muscle sarcomeres with a deletion in titin.

The giant protein titin regulates the length of the striated muscle thick filament.

Effect of levosimendan on the contractility of muscle fibers from nemaline myopathy patients with mutations in the nebulin gene.

Detyrosinated microtubules modulate mechanotransduction in heart and skeletal muscle.

Computing Average Passive Forces in Sarcomeres in Length-Ramp Simulations .

The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms.

Reduced passive force in skeletal muscles lacking protein arginylation.

Pompe disease results in a Golgi-based glycosylation deficit in human induced pluripotent stem cell-derived cardiomyocytes.

Alterations at the Cross-Bridge Level Are Associated with a Paradoxical Gain of Muscle Function In Vivo in a Mouse Model of Nemaline Myopathy

Human myocytes are protected from titin aggregation-induced stiffening by small heat shock proteins

A HaloTag-TEV genetic cassette for mechanical phenotyping of proteins from tissues

Disruption of cardiac thin filament assembly arising from a mutation in LMOD2: A novel mechanism of neonatal dilated cardiomyopathy

Fast skeletal muscle troponin activator CK-2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Myosin filament sliding through the Z-disc relates striated muscle fibre structure to function

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Protein phosphatase 5 regulates titin phosphorylation and function at a sarcomere-associated mechanosensor complex in cardiomyocytes

Reversible temporally-specific inhibition of muscle using a light-activated chloride channel

Myosin Head Configurations in Resting and Contracting Murine Skeletal Muscle

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Deleting Full Length Titin Versus the Titin M-Band Region Leads to Differential Mechanosignaling and Cardiac Phenotypes.

Decreased force enhancement in skeletal muscle sarcomeres with a deletion in titin.

The giant protein titin regulates the length of the striated muscle thick filament.

Effect of levosimendan on the contractility of muscle fibers from nemaline myopathy patients with mutations in the nebulin gene.

Detyrosinated microtubules modulate mechanotransduction in heart and skeletal muscle.

Computing Average Passive Forces in Sarcomeres in Length-Ramp Simulations .

The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms.

Reduced passive force in skeletal muscles lacking protein arginylation.

Pompe disease results in a Golgi-based glycosylation deficit in human induced pluripotent stem cell-derived cardiomyocytes.

Alterations at the Cross-Bridge Level Are Associated with a Paradoxical Gain of Muscle Function In Vivo in a Mouse Model of Nemaline Myopathy

Human myocytes are protected from titin aggregation-induced stiffening by small heat shock proteins

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