Myopathy Archives

Silencing of the Ca2+ Channel ORAI1 Improves the Multi-Systemic Phenotype of Tubular Aggregate Myopathy (TAM) and Stormorken Syndrome (STRMK) in Mice

A HaloTag-TEV genetic cassette for mechanical phenotyping of proteins from tissues

Fast skeletal muscle troponin activator CK-2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

Disruption of cardiac thin filament assembly arising from a mutation in LMOD2: A novel mechanism of neonatal dilated cardiomyopathy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Myosin filament sliding through the Z-disc relates striated muscle fibre structure to function

Protein phosphatase 5 regulates titin phosphorylation and function at a sarcomere-associated mechanosensor complex in cardiomyocytes

Reversible temporally-specific inhibition of muscle using a light-activated chloride channel

Myosin Head Configurations in Resting and Contracting Murine Skeletal Muscle

Deleting Full Length Titin Versus the Titin M-Band Region Leads to Differential Mechanosignaling and Cardiac Phenotypes.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Effect of levosimendan on the contractility of muscle fibers from nemaline myopathy patients with mutations in the nebulin gene.

The giant protein titin regulates the length of the striated muscle thick filament.

Decreased force enhancement in skeletal muscle sarcomeres with a deletion in titin.

Reduced passive force in skeletal muscles lacking protein arginylation.

The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms.

Computing Average Passive Forces in Sarcomeres in Length-Ramp Simulations .

Detyrosinated microtubules modulate mechanotransduction in heart and skeletal muscle.

Muscle-generated BDNF is a sexually dimorphic myokine that controls metabolic flexibility

Inhibition of sphingolipid de novo synthesis counteracts muscular dystrophy

Silencing of the Ca2+ Channel ORAI1 Improves the Multi-Systemic Phenotype of Tubular Aggregate Myopathy (TAM) and Stormorken Syndrome (STRMK) in Mice

A HaloTag-TEV genetic cassette for mechanical phenotyping of proteins from tissues

Fast skeletal muscle troponin activator CK-2066260 increases fatigue resistance by reducing the energetic cost of muscle contraction

Disruption of cardiac thin filament assembly arising from a mutation in LMOD2: A novel mechanism of neonatal dilated cardiomyopathy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Downsizing the molecular spring of the giant protein titin reveals that skeletal muscle titin determines passive stiffness and drives longitudinal hypertrophy

Myosin filament sliding through the Z-disc relates striated muscle fibre structure to function

Protein phosphatase 5 regulates titin phosphorylation and function at a sarcomere-associated mechanosensor complex in cardiomyocytes

Reversible temporally-specific inhibition of muscle using a light-activated chloride channel

Myosin Head Configurations in Resting and Contracting Murine Skeletal Muscle

Deleting Full Length Titin Versus the Titin M-Band Region Leads to Differential Mechanosignaling and Cardiac Phenotypes.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Slow-twitch skeletal muscle defects accompany cardiac dysfunction in transgenic mice with a mutation in the myosin regulatory light chain.

Cardiac myosin binding protein-C phosphorylation regulates the super-relaxed state of myosin.

Effect of levosimendan on the contractility of muscle fibers from nemaline myopathy patients with mutations in the nebulin gene.

The giant protein titin regulates the length of the striated muscle thick filament.

Decreased force enhancement in skeletal muscle sarcomeres with a deletion in titin.

Reduced passive force in skeletal muscles lacking protein arginylation.

The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms.

Computing Average Passive Forces in Sarcomeres in Length-Ramp Simulations .

Detyrosinated microtubules modulate mechanotransduction in heart and skeletal muscle.

Recent Posts

Categories

Archives