Cardiovascular Archives

Timing and magnitude of systolic stretch affect myofilament activation and mechanical work.”

Cold acclimation increases cardiac myofilament function and ventricular pressure generation in trout.

Myofilament dysfunction contributes to impaired myocardial contraction in the infarct border zone.

Automated analysis of contractile force and Ca2+ transients in engineered heart tissue.

A gain-of-function mutation in the M-domain of cardiac myosin-binding protein-C increases binding to actin

Diaphragm atrophy and contractile dysfunction in a murine model of pulmonary hypertension.

Ablation of cardiac myosin-binding protein-C accelerates contractile kinetics in engineered cardiac tissue.

Doxorubicin induced myocardial injury is exacerbated following ischaemic stress via opening of the mitochondrial permeability transition pore.

Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction.

Contractility of myofibrils from the heart and diaphragm muscles measured with atomic force cantilevers: Effects of heart-specific deletion of arginyl-tRNA-protein transferase.

Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction.

Ablation of cardiac myosin-binding protein-C accelerates contractile kinetics in engineered cardiac tissue.

Diaphragm weakness in pulmonary arterial hypertension: role of sarcomeric dysfunction.

Diaphragm weakness in pulmonary arterial hypertension: role of sarcomeric dysfunction.

Diaphragm muscle fiber weakness in pulmonary hypertension.

Fabrication and evaluation of reconstructed cardiac tissue and its application to bio-actuated microdevices.

Fabrication and evaluation of reconstructed cardiac tissue and its application to bio-actuated microdevices.

Timing and magnitude of systolic stretch affect myofilament activation and mechanical work.”

Cold acclimation increases cardiac myofilament function and ventricular pressure generation in trout.

Myofilament dysfunction contributes to impaired myocardial contraction in the infarct border zone.

Automated analysis of contractile force and Ca2+ transients in engineered heart tissue.

A gain-of-function mutation in the M-domain of cardiac myosin-binding protein-C increases binding to actin

Diaphragm atrophy and contractile dysfunction in a murine model of pulmonary hypertension.

Ablation of cardiac myosin-binding protein-C accelerates contractile kinetics in engineered cardiac tissue.

Doxorubicin induced myocardial injury is exacerbated following ischaemic stress via opening of the mitochondrial permeability transition pore.

Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction.

Contractility of myofibrils from the heart and diaphragm muscles measured with atomic force cantilevers: Effects of heart-specific deletion of arginyl-tRNA-protein transferase.

Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction.

Ablation of cardiac myosin-binding protein-C accelerates contractile kinetics in engineered cardiac tissue.

Diaphragm weakness in pulmonary arterial hypertension: role of sarcomeric dysfunction.

Diaphragm weakness in pulmonary arterial hypertension: role of sarcomeric dysfunction.

Diaphragm muscle fiber weakness in pulmonary hypertension.

Fabrication and evaluation of reconstructed cardiac tissue and its application to bio-actuated microdevices.

Fabrication and evaluation of reconstructed cardiac tissue and its application to bio-actuated microdevices.

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